Cock van oosterhout

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Use the link below to share a full-text version of this article with your friends and colleagues. Learn more. Metapopulations were maintained for seven generations during which coancestries and pedigrees were established.

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The co-evolutionary arms race between host immune genes and parasite virulence genes is known as Red Queen dynamics. Such observations are often taken as evidence of host-parasite co-evolution. Here, we use computer simulations of the Major Histocompatibility Complex MHC of guppies Poecilia reticulata to study the turnover rate of alleles temporal genetic differentiation, G' ST.

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Cock van Oosterhout PhD Prof. Predation pressure can alter the morphology, physiology, life history, and behavior of prey; this in turn can change how surviving prey interact with parasites. These trait-mediated indirect effects may change in direction or intensity during growth or, in sexually dimorphic species, between the sexes.

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The original article was published in Genome Biology 18 After publication of this article [ 1 ] we noticed that reference 50 was incorrect. The correct reference 50 is as follows:.

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All publications more feeds Perspectives in Phycology Vol. DOI:

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Cock van Oosterhout did his PhD on captive metapopulations of the butterfly Bicyclus anynana at Leiden University in the Netherlands under the supervision of Paul Brakefield. He received an NERC Fellowship testing conservation genetic breeding protocols, reintroduction and genetic supplementation programs using guppies as an empirical model and did this research in Hull and Trinidad. He developed software applications, including Micro-Checker for population genetic analysis of microsatellites, as well as models of host-parasite coevolution and population genetic simulations, and more recently, he designed software for whole genome analysis of recombination Hybrid-Check.

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How generalist parasites with wide host ranges can evolve is a central question in parasite evolution. Albugo candida is an obligate biotrophic parasite that consists of many physiological races that each specialize on distinct Brassicaceae host species. Sequential infection experiments show that infection by adapted races enables subsequent infection of hosts by normally non-infecting races.

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